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LAH #579.pdf

lah579_cysdef.pdf
Preview of LAH #579
🔗 Source: herzenberglab.stanford.edu
📊 Size: 338 KB
👤 Author: 300505
⬇️ Downloads: 73

Summary

Cysteine/Glutathione deficiency is a significant and treatable corollary of disease. Glutathione (GSH) depletion occurs when cysteine supplies are inadequate, leading to oxidant injury. N-Acetylcysteine (NAC) is converted to cysteine, replenishing GSH. 102 randomized placebo-controlled trials (RPCTs) have examined NAC's effects in various diseases, with 72 reporting beneficial effects, suggesting cysteine/GSH deficiency contributes to disease pathophysiology and NAC may be a useful therapeutic adjunct. GSH is a central component of the oxidative-reductive apparatus, detoxifying reactive oxygen species and other molecules, and its depletion must be continually replenished to maintain cell viability.

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Description

Cysteine/Glutathione deficiency is a significant and treatable corollary of disease.

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  • File Format: PDF
  • File Size: 338 KB
  • Pages: 38
  • Language: EN
  • Author: 300505
  • Total Downloads: 73
  • Last Updated: 1 week ago

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